Hyphema

 

Hyphema: Clinical Essentials

Ophthalmology High-Yield: Trauma & Emergencies

1. Causes

  • Traumatic (Most Common): Blunt ocular trauma causing rupture of iris/ciliary body vessels.
  • Spontaneous: Neovascularization (Rubeosis iridis in PDR), iris tumors (e.g., juvenile xanthogranuloma), or clotting disorders.

2. Clinical Features (C/F)

Feature Description
Signs Blood in the anterior chamber (leveling effect).
Symptoms Pain, photophobia, blurred vision.
Complications Secondary glaucoma (blocked TM), corneal blood staining, and re-bleeding.

3. Management (Rx)

  • Medical (Standard): Bed rest (head elevated 30-45°), eye shield, topical cycloplegics (to rest the iris), and topical corticosteroids (to reduce inflammation).
  • IOP Management: If pressure is elevated, use beta-blockers or alpha-agonists. Avoid miotics (can increase inflammation) and avoid aspirin/NSAIDs (risk of re-bleeding).
  • Surgical: Indications for anterior chamber paracentesis/washout:
    • Uncontrolled IOP despite maximal medical therapy.
    • Corneal blood staining.
    • Total hyphema persisting > 5 days.

4. NEET PG High-Yield Pearls

  • “Eight-Ball Hyphema”: Total black/dark red hyphema; indicates total anterior chamber filled with clotted blood; extreme risk for secondary glaucoma.
  • Sickle Cell Trait: Mandatory screening; even small hyphemas can cause massive IOP spikes due to sickling of RBCs in the trabecular meshwork.
  • Re-bleed: Most likely to occur 3–5 days post-injury as the initial clot retracts.