U01.11.061 Seborrheic dermatitis

 

Learning Objectives

Identify the classic clinical features of Seborrheic Dermatitis, including its characteristic “greasy” appearance and predilection for sebaceous-rich areas. Master the systemic associations, specifically HIV and Parkinson’s disease, and understand the role of Malassezia spp. in its pathogenesis.


1. Clinical Characteristics & Presentation

Seborrheic dermatitis is a common inflammatory condition that affects areas of the body with a high density of oil-producing glands. It presents differently across age groups but maintains a consistent “greasy” morphology.

Feature Description High-Yield Color/Texture
Morphology Erythematous, well-demarcated plaques. Greasy yellow scales.
Distribution Scalp (dandruff), eyebrows, chest, and nasolabial folds. Areas rich in sebaceous glands.
Infant Form Known as “Cradle Cap”. Usually self-limiting.

2. Pathophysiology & Systemic Associations

While the exact cause is not fully understood, the condition is strongly linked to the presence of skin flora and specific underlying neurological or immunological states.

Category Associated Factor Clinical Context
Microbiology Malassezia spp. A yeast-like fungus that thrives on sebum.
Neurological Parkinson Disease Increased incidence and severity of dermatitis.
Immunological HIV Infection An extensive or “explosive” onset may be a sentinel sign of HIV.

3. Management & Treatment

Treatment focuses on reducing the fungal load and calming the associated inflammation.

Therapy Type Action
Topical Antifungals Target the Malassezia (e.g., ketoconazole).
Topical Glucocorticoids Reduce inflammation and erythema.

Activity: Clinical Correlations

High-Yield Mnemonics:

  • Seborrheic = Sebaceous (oil) glands + Scales (yellow/greasy).
  • HIV & Parkinson: If a patient has unusually severe dandruff or facial redness, check for HIV or Parkinson’s.
  • Malassezia: This is the same fungus associated with Tinea Versicolor.

Activity: