U01.01.087 Protein-energy malnutrition

 

Learning Objectives

  • Distinguish between Kwashiorkor and Marasmus based on clinical presentation.
  • Explain the pathophysiology of edema and fatty liver in protein malnutrition.
  • Identify the “MEALS” mnemonic for rapid board recall.

1. Kwashiorkor (Protein Malnutrition)

Kwashiorkor is primarily a protein deficiency in the setting of adequate (or slightly deficient) caloric intake. It is classically seen in a child after being weaned from breast milk to a high-carbohydrate, protein-poor diet.

  • Edema: Caused by a decrease in plasma oncotic pressure due to low serum albumin. This is further exacerbated by increased antidiuretic hormone (ADH).
  • Fatty Liver (Hepatomegaly): Malnutrition leads to decreased apolipoprotein synthesis. Without these proteins, lipids cannot be transported out of the liver, resulting in fatty change.
  • Clinical Appearance: Small child with a characteristically swollen abdomen.

Mnemonic: Kwashiorkor results from protein-deficient MEALS:

  • Malnutrition
  • Edema
  • Anemia
  • Liver (Fatty)
  • Skin lesions (e.g., hyperkeratosis, dyspigmentation)

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2. Marasmus (Total Caloric Malnutrition)

Marasmus is a state of overall energy deficiency. The diet is deficient in total calories, but no specific nutrients are absent.

  • Muscle Wasting: The body utilizes muscle tissue for energy, leading to significant emaciation.
  • Absence of Edema: Unlike Kwashiorkor, oncotic pressure is relatively maintained, so there is no edema.
  • Growth: Linear growth is often maintained in acute cases, whereas chronic malnutrition eventually stunts linear growth.

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Summary Comparison

Feature Kwashiorkor Marasmus
Primary Deficiency Protein Total Calories (Energy)
Edema Present Absent
Liver Involvement Fatty Liver (Apolipoprotein ↓) Uncommon
Body Habitus Swollen abdomen (“Potbelly”) Emaciated (“Skin and bones”)

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